Everything about Myocardial Rupture totally explained
Myocardial rupture is a laceration or tearing of the walls of the
ventricles or
atria of the
heart, of the
interatrial or
interventricular septum, of the
papillary muscles or
chordae tendineae or of one of the
valves of the heart. It is most commonly seen as a serious sequelae of an acute
myocardial infarction (heart attack).
Etiology
The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. Other causes of rupture include cardiac trauma,
endocarditis (infection of the heart),
cardiac tumors, infiltrative diseases of the heart, and delay of revascularization greater than 2 hours. On the other hand, if
primary percutaneous coronary intervention is performed to abort the infarction, the incidence of rupture is significantly lowered. The incidence of myocardial rupture if PCI is performed in the setting of an acute myocardial infarction is about 1 percent.
Classification
Myocardial ruptures can be classified as one of three types. Type I myocardial rupture is an abrupt slit-like tear that generally occurs within 24 hours of an acute myocardial infarction. Type II is an erosion of the infarcted myocardium, which is suggestive of a slow tear of the dead myocardium. Type II ruptures typically occur more than 24 hours after the infarction occurred. Type III ruptures are characterized by early aneurysm formation and subsequent rupture of the aneurysm.
Another method for classifying myocardial ruptures is by the anatomical portion of the heart that has ruptured. By far the most dramatic is rupture of the free wall of the left of right ventricles, as this is associated with immediate hemodynamic collapse and death secondary to acute
pericardial tamponade. Rupture of the interventricular septum will cause a
ventricular septal defect. Rupture of a papillary muscle will cause acute
mitral regurgitation.
Signs and symptoms
Symptoms of myocardial rupture are recurrent or persistent
chest pain,
syncope, and distension of
jugular veins.
Diagnosis
Due to the acute hemodynamic deterioration associated with myocardial rupture, the diagnosis is generally made based on physical examination, changes in the vital signs, and clinical suspicion. The diagnosis can be confirmed with
echocardiography.
Treatment
The treatment for myocardial rupture is supportive in the immediate setting and surgical correction of the rupture, if feasible. A certain small percentage of individuals don't seek medical attention in the acute setting and survive to see the physician days or weeks later. In this setting, it may be reasonable to treat the rupture medically and delay or avoid surgery completely, depending on the individual's
comorbid medical issues.
Prognosis
The prognosis of myocardial rupture is dependant on a number of factors, including which portion of the myocardium is involved in the rupture. In one case series, if myocardial rupture involved the free wall of the
left ventricle, the mortality rate was 100 percent.
Even if the individual survives the initial hemodynamic sequelae of the rupture, the 30 day mortality is still significantly higher than if rupture didn't occur.
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